Insulin, Leptin, and Blood Sugar Why Diabetic Medication Fails
by Byron Richards, Health Freedom Editor

AMERICA, LAND OF the CHUNKY TEENS. -  Type II diabetes is a  metabolic problem, simple to cure or 'rearrange' and eliminate as a hazard..However, BEING CHUBBY, overly oral and disposed to the disease is such a national habit or mind set, (especially in a world riddled with famine!) Newest TREND is  -- many of our young people are becoming type II diabetic and dying of it. I wonder if those emaciated third worlders look on at the statistics and grimly enjoy the fact that we are EATING ourselves to death while they are STARVING themselves in the same direction, but not by their own WILL has any irony? So in an increasingly OLIGARCHIC EMPIRE, It is fitting that the FAT DIE FIRST! Scientists who test lab rats,  find that the STARVING ones live longer. However, it is also a national disgrace that millions of type II diabetic patients are being injured with commonly used diabetic medications --- costly PHARM known to make their metabolic situation worse. So we have to do everything we can to prevent diabetes. SOME signs that you are getting type 2 adult onset diabetes:

a.) longtime hypoglycemia meaning munchies, even interrupting sleep, and
that is your history for a lot of years. You have fat middle.

b.) suddenly munchies stop dead. You're just congratulating self when U

c.) numbness in feet when u awake after long inactivity meaning you had
probable high blood sugar.

d.) dizziness, vision not so good.

Find herbs to treat pancreas.(see below) Drink more water to dilute sugar in blood after sweets, and then immediately do more activity within the hours after eating fruits/sweets.

LOSE that extra WEIGHT. SEE THE SOLAR DIET. Switch away from all carbs to prots and veggies. Chow down so heavily on these that you have no room for potatoes, rice, bread which should not even be in the house.

An overwhelming body of science demonstrates that insulin resistance  leads to obesity and vice versa. Once this problem sets in a person heads down a path of ever-worsening metabolic control as diabetes-related issues, cholesterol problems, and heart disease risk factors pile up. If nothing is done, very poor health and early death are certain. Look at chubby Tim Russert who died recently, at a very young age. Of heart problems.

However, the Big Pharma blood-sugar remedies turn out to be really bad for health and actually complicate rather than improve the patient's health. Even when the drugs aren't directly damaging in a major way, they fail to address the actual reasons for diabetes and typically have the net result of making the factors causing diabetes worse. I know that may seem hard to believe -- but it is true, and I will explain it shortly.

On December 17, 2008 the New England Journal of Medicine put the nail in the coffin on another dismal year for the theory of drugs to treat disease, reporting that aggressive use of blood-sugar-lowering medication to prevent heart disease was a complete failure. Its not that lowering blood sugar in this patient population didn't do anything: it made the patients heavier and more hypoglycemic. This newer study followed equally dismal results from the ACCORD trial (Action to Control Cardiovascular Risk in Diabetes), which earlier in 2008 found a 22% increased rate of death in diabetic patients who were aggressively treated with medications.

Some of the newer diabetes medications like Avandia are quite deadly and likely to injure in multiple ways (such as doubling the risk for bone fractures). Scientists at the FDA were so concerned this drug would cause heart failure that they wanted a black box warning on it from the start. However, Von Eschenbach and his band of FDA management goons forced FDA scientists to not warn anyone! As Avandia-treated bodies starting showing up on the doorsteps of morgues around the country, Congress started asking questions. Eventually scientists reported a 43% increase in the risk for heart failure from Avandia; however, the FDA had this data from the start and didn't tell anyone. The FDA allowed Avandia sales to reach 3.2 billion per year while killing and injuring a lot of patients. Even when the high-profile type II diabetic 'chubbers'  Tim Russert keeled over dead from a heart attack, nobody in the media seemed interested to know if he was taking Avandia. He surely WAS. No way he wasn't a DIAB II

MEANWHILE BACK AT THE RANCH! At the same time that the FDA was helping to create a market for deadly Avandia sales, they sent out twenty-four warning letters to small dietary supplement companies telling them that their promotion of various products to lower blood sugar, correct insulin resistance, or improve diabetes is against the law. Against whose law? Certainly not the first amendment. Oh, that EUROPEAN CODEX thing they're always trying to push past the United Nations so it'll become WORLD LAW? That's some new reg that no supplement or vitamin is allowed! HOPE that doesn't make it! PRAY that it doesn't!

The Blood Sugar Con Job The FDA and Big Pharma get away with this nonsense because they set the standard for drug effectiveness on a surrogate endpoint or biomarker, in this case the blood sugar level. Their logic is that if blood sugar levels are better than health must be better. This means that any drug that takes a toxic sledgehammer to blood sugar levels, knocking them down, is just fine in the eyes of the FDA even though the drug leaves a trail of damage around the body -- even killing the person!

On a lesser scale, but using similar logic, doctors think that any
medication that helps lower blood sugar is doing the patient a favor.
Never do they ask the most obvious question: If you are lowering blood
sugar with a drug where is the sugar going? The answer is: most often to
stored fat. Or the sugar is simply forced into cells and kills them
because the cells couldn't use the sugar.

What is a diabetes patient to do? And how does anyone whose blood sugar
is beginning to rise get the problem under control? Answering these
questions requires that you understand something about the subject, as
it is rather obvious most doctors, even those who treat diabetes
patients as their primary business, don't have adequate practical
knowledge to fix the great majority of people coming to them for help.

Blood Sugar 101 First off, your blood is not a very sweet beverage.
Normal fasting blood sugar is slightly less than one teaspoon of sugar
in your five or so quarts of blood. What happens when you drink a
sugar-sweetened Coke that contains ten teaspoons of sugar?

When you eat any food, even fat, your insulin level will rise. Higher
amounts of refined carbohydrates or simple sugars will raise your
insulin faster and in higher amounts. The greater the fiber content of
your diet, the slower insulin is raised and the more controlled the
process. When you eat a large meal, regardless of the type of calories,
it causes a large and difficult to manage surge in insulin.

Insulin is a taxicab for calories. Its goal is to take blood sugar, as
its passenger, to various locations in your body that want it. It helps
if you are active, as some of the sugar is more likely to be wanted by
cells in your body, including your many muscle cells.

Blood sugar is fuel, like gasoline is to a car. Your brain must have a
regular supply or your head conks out. Thus, following a meal your
insulin taxis are busy transporting sugar through your circulation and
out to your cells, hoping to find cells that need some sugar.

In a healthy person, insulin drops off a whopping sixty percent of the
sugar at your liver  which acts as a warehouse, converting the blood
sugar to glycogen for storage.

Insulin is released by your pancreas in two phases. The first phase is
from insulin that is already made and stored in your pancreas, which is
just waiting for some food to come along. This is your first wave of
taxis coming to pick up the first set of blood-sugar passengers. The
release of this insulin triggers your pancreas's beta cells to start
making more insulin to deal with the rest of the meal.

As you are eating, some of the insulin transports blood sugar to your
white adipose tissue or stored fat. The blood sugar is taken up by fat
cells, activating their metabolism, in turn producing the hormone
leptin. Leptin now enters your blood and begins traveling up to your
brain. The more you eat, the more insulin you make, and the more leptin
you make.

When leptin levels get high enough, meaning you have eaten enough, then
leptin permeates into your brain and tells your subconscious brain you
are full. At the same time the higher levels of leptin are also telling
your pancreas that you are full, which turns off the beta cell
production of insulin, as no more taxis are needed.

If you ate the right amount of food for your physical activity level
then blood sugar always has some place healthy to go, insulin rises and
falls in a controlled manner, as does leptin.

When insulin has too many blood sugar passengers and cells don't need
any sugar, then insulin stimulates the production of triglycerides
(which can become stored fat). This is how you gain weight.
Unfortunately, as triglycerides elevate in your blood they get in the
way of leptin getting into your brain. This keeps you eating more than
you need to because you don't have a full signal yet, a problem called
leptin resistance. This encourages even further insulin-driven
triglyceride formation, making it more likely you will gain weight.

If you stop eating so much and start exercising more, then this
simple-case issue can improve and will often bounce back to normal
function -- thus the basic idea of eating less and exercising more to
lose weight.

If you continually eat too much and are gaining weight, then cells get

tired of seeing insulin taxis driving up. In fact, they shutter their
windows and lock their doors, insulin becomes persona non grata. The
reason for this rejection of insulin is rather simple if the cells
took in blood sugar when they can't use it, because they already have
enough, then the extra sugar will caramelize and kill the cell.
Rejecting insulin is a self defense measure. This is the mechanism
behind basic insulin resistance at the cellular level.

If this problem keeps going, blood sugar levels continue to rise,
insulin resistance gets worse, leptin resistance gets worse, cholesterol
levels go up, blood pressure goes up, triglyceride levels go up, and
inflammation really starts heating up. Eventually this leads to type II
diabetes, along with many risk factors for heart disease, and then heart
and kidney disease lock into place.

The problem for any kind of blood sugar medication is that it only
addresses one of many mechanisms that aren't working right, while
creating its own side effect complications. At best, it is a temporary
solution with a narrow scope of benefit - and does not address the true
source of the problem.

Furthermore, when more drugs are added in an effort to more
comprehensively address the multiple aspects of the problem, then side
effects really pile up in a hurry and injure the patient. This means the
risk of multiple drug treatment far outweighs the benefits, even though
one or two drugs can't get the job done.

More often, the medication either forces sugar into cells †killing or
injuring them or it transfers the sugar into fat, making leptin problems
worse that in turn make insulin problems almost impossible to solve. The
shortcoming of these medications is openly acknowledged in the
scientific literature, yet this is what passes for standard medical
care. It is a disgrace.

Basic Diet Adjustments for Insulin Improvement

The absolute worst possible dietary pattern of eating for a type II diabetic IS THE
FOR ALMOST ALL TYPE II DIABETIC PATIENTS, helping to lock in a national
epidemic of type II diabetes. They routinely tell individuals to snack
in order to maintain their blood sugar levels and to "stoke" their
metabolism with fuel.

In normal health, when you haven't eaten for three hours, insulin levels
return to a baseline. Now your pancreas makes a different hormone called
glucagon. This hormone tells your liver to release the sugar it has
stored (glycogen) to sustain your blood sugar levels, and as it does
this it turns on your liver's fat burning system. Thus, under the
influence of glucagon your liver simultaneously uses sugar and fat to
sustain your blood sugar a true fat burning time that helps to clear
up stagnating levels of triglycerides in your blood.

If you snack on anything surpassing thirty calories you will raise
insulin, which automatically turns off glucagon, causes fat burning to
stop, and blunts the use of sugar that has been stored in your liver.
However, since you haven't used the stored sugar in your liver, then
insulin can't put more sugar back in your liver as it normally would,
meaning it will readily turn blood sugar into fat (even if you snacked
on something with no fat).

You are supposed to get a snack between meals but it is supposed to
come from your liver, not from eating.

The worst things for leptin and insulin are eating between meals, eating
large meals, eating low fiber, eating high refined sugar or refined
carbohydrates, not eating enough quality protein, and not exercising.

If you eat anything after dinner you make matters even worse, because
now you reduce the optimal access into your stored fat during sleep, a
prime opportunity to burn fat.

When this system is abused and weight is gained, then fat begins to
accumulate in excess in your liver. The fat clogs your liver's
metabolism and reduces the ability of sugar to store in your liver
following a meal. This is liver insulin resistance caused by fatty build
up. This means that you are much more likely to become hypoglycemic or
low blood sugar between meals as you don't have enough sugar in your
warehouse to use for blood sugar between meals.

This same fatty liver problem also gets in the way of how glucagon would
burn fat between meals, causing glucagon to synthesize sugar in an
inappropriate and out-of-control manner, making blood sugar go high even
though you haven't eaten. This is why diabetics wake up with very high
fasting blood sugar levels. These are complicated metabolic problems
that are more difficult to fix than simple case insulin resistance.

Furthermore, your pancreas starts to tune out leptin, meaning that
leptin resistance is occurring at the level of beta cells and the beta
cells aren't getting the leptin message to stop making insulin in a
timely manner. This causes extra insulin to be made, which excessively
lowers blood sugar by turning sugar to fat, while simultaneously
inducing hypoglycemia or low blood sugar symptoms. This makes a person
want to eat again two to three hours after the previous meal, in turn
making the whole problem worse.

And it's about this time, with metabolism clogged and broken, that a
young overweight person goes to the doctor and finds out he or she is
type II diabetic.

If the problem continues, then inflammation begins to damage the
insulin-secreting beta cells. Not only are these beta cells now leptin
resistant, causing them to overproduce insulin and get tired out, but
they are getting damaged and their numbers are declining †meaning now
they can't make insulin either. This sends a person down a path of a
mixture of type I and type II diabetes - with an autoimmune component
sometimes thrown in for good measure, a problem that is seen
progressively more often in today's older type II diabetics.

The Complexity of Insulin and Leptin Problems Well, if you thought that
was hard to understand, then realize that the previous description was
the rather simple explanation of the problem (and worth reading over
again until you understand it). The metabolic problems of a diabetic
patient are actually far more complex. To prevent yourself from becoming
diabetic or to get over the problem, it is very important to understand
even more information.

As your fat cells expand and cram into each other, a highly inflammatory
state occurs within your white adipose tissue. These inflammatory
signals aggravate and lock in the various problems discussed in previous
sections. Another hormone made in fat, adiponectin, is a major player in
this equation.

In health, leptin and adiponectin elevate in harmony, side by side. When
leptin resistance occurs, which is common anytime someone starts gaining
weight, then adiponectin levels begin to fall. The fall in adiponectin
is caused by the inflammation in white adipose tissue. Interestingly,
adiponectin is a primary anti-inflammatory hormone within white adipose
tissue. Thus, the inflammation of progressive weight gain eventually
overwhelms adiponectin, at which point serious problems really set in.

Once adiponectin levels fall too much, then inflammation in fat cells
really ramps up. The greater the weight gain and leptin resistance, the
farther adiponectin levels fall. When adiponectin levels fall your
liver's ability to process sugar and insulin dramatically worsen, and
your muscles become resistant to insulin †speeding the onset of type
II diabetes.

And that's not all †your brain becomes insulin resistant which is
actually caused by the leptin resistance, and this problem is now found
to lock in all problems of insulin resistance around your body. A great
deal of research indicates that the failure of insulin and leptin to
register properly in your brain, along with falling adiponectin levels,
creates a highly inflammatory state of affairs that is the prime cause
of worsening blood sugar regulation and eventual type II diabetes.

Diabetic medication does not address these issues, and often makes them
worse over time ---even if blood sugar numbers appear better for a
period of time. Treating blood sugar numbers is not treating the cause
of anything. It's like saying the cause of a house fire is because the
Fire Department didn't show up fast enough so now let's put a fire
hydrant in everyone's house so we can put out fires faster. That is an
accurate analogy of how the Big Pharma-trained medical profession
manages the type II diabetic population in our country. Even worse,
their monopoly and inept care is sanctioned by the FDA and FTC as law,
who act as police force bullies to stamp out any competing interests.

How to Solve the Diabetes Problem There is no quick fix for type II
diabetes, or even fasting blood sugar levels that are on the rise. To be
healthy, your fasting blood sugar should never be above 90. However,
real health is determined by achieving this number because your body is
working properly.

There is no vitamin, mineral, or miracle pill that can automatically
prevent or treat diabetes. What you are trying to do is create a
nutritional environment that supports your body working normally.

Individuals with simple-case insulin and leptin resistance, who cut back
on junk, exercise more, make appropriate dietary adjustments, and use
some basic dietary supplement support can typically bring these smaller
problems in line relatively quickly.

When problems are more serious they are complicated by the fact that the
liver, pancreas, white adipose tissue, circulatory system, and
subconscious brain have been irritated and even damaged by inflammatory
compounds. This fact alone takes time to heal †it is not a simple
nutritional deficiency †it is more like a badly sprained ankle.

Additionally, the liver, muscles, and circulation are clogged with
accumulating fat. This is like trying to cook a meal in a filthy
kitchen, it is going to be challenging. This problem will only gradually
improve as weight is lost, it never improves until the weight is lost,
and it is always getting worse if weight is being gained.

The solution is to create a pattern of health, rooted in a good diet and
exercise, wherein these more advanced problems can begin to correct
themselves. Type II diabetes is completely reversible for almost
everyone if only diabetics knew what to do.

Dietary Basics to Correct Type II Diabetes Dietary fiber, especially
soluble fiber, is critical for the repair of blood sugar problems.
Soluble fiber is vital for slowing the rate at which sugar enters your
blood, thus enabling your insulin system to function with less stress.
It also helps you maintain healthier levels of triglycerides and
cholesterol. Individuals struggling with blood sugar issues need 35 to
50 grams of fiber per day, much of it as soluble fiber (a higher level
than the 25 grams the government recommends for general health). Higher
amounts are also very helpful to curb appetite and support weight loss.

A serving of fruit or vegetables or a cup of oatmeal average about 4
grams of soluble fiber per serving. Legumes have about 8 grams of
soluble fiber per serving (avoid soy). It is really easy to get higher
amounts of soluble fiber with high quality fiber drinks. Pysllium, oat
beta glucan concentrates, arabinogalactan, and partially hydrolyzed guar
gum are just a few of the many fiber options available for consumers to
easily boost soluble fiber intake.

Protein is vital in the repair of blood sugar metabolism. A minimal
objective for protein intake is three-fourths of your ideal weight in
grams of protein per day. To improve insulin function, the key proteins
that help are rich in branch chain amino acids especially leucine.
Unlike any other amino acids, leucine directly communicates to insulin,
instructing it to work efficiently in muscle. This not only helps
preserve your muscle mass as you lose weight, it helps your muscles use
glucose as fuel, in turn supporting healthy insulin function. Whey
protein also helps leptin enter your brain more efficiently, supporting
normal insulin function in your nervous system.

The highest sources of leucine-containing foods are animal and dairy
sources. Cottage cheese and red meat top the list; other sources include
milk, cheese, eggs, pork, fish, chicken, legumes, peanuts, nuts, and
seeds. If you avoid red meat and dairy products, it is harder it is to
get leucine-containing foods in higher amounts †though not
impossible. Using whey protein makes it easy. I always recommend
individuals stay away from processed soy protein (like soy protein
drinks), as it is anti-thyroid in higher amounts.

The key foods to eliminate are refined carbohydrates, refined sugar, and
high fructose corn syrup. Complex carbohydrates and fruit should be
eaten at meals, several servings of each per day. Saturated fat and
cholesterol-containing foods do not need to be avoided just don't
eat them in excess. A bite or two of dessert is permissible at a dinner
meal, but never between meals or as a snack. Avoid adding any sweetener
to food, whether natural or artificial, as they skew your taste buds to
be addicted to food in larger amounts than you truly need. Soda drinks
should be completely avoided. Coffee or tea in moderation. Eat organic
whenever possible. Raw foods and vegetable juicing is great †go easy
on the carrots and apples/fruit. The Five Rules of the Leptin Diet

As I have explained in my books, Mastering Leptin and The Leptin Diet,
there are five basic rules for eating that help correct leptin and
insulin problems. They are:

Rule 1: Never eat after dinner. Rule 2: Eat three meals a day (do not
snack). Rule 3: Do not eat large meals. Rule 4: Eat a breakfast
containing protein. Rule 5: Reduce the amount of carbohydrates eaten.

While these rules are fairly simple, they are backed by considerable
cutting-edge science. They help you extract more energy from less food.
They form the foundation for a healthy style of eating that you can
follow for a lifetime without any feeling of deprivation.

Some individuals who are type II diabetic may struggle a bit to go five
hours between meals or make it through the night. This does not mean the
rules are wrong, it simply means that liver and pancreatic fitness need
to be restored. You can eat four smaller meals four hours apart to get
started, or gradually implement rules until you can do them all. As you
improve, work towards the three meals a day. You will be surprised how
easy and effective it actually is. Numerous type II diabetic patients
have made dramatic improvement by following the Leptin Diet.

How Dietary Supplements Can Help Correct Type II Diabetes Many people
will be able to correct type II diabetes simply by increasing exercise,
following the Five Rules of The Leptin Diet, and improving the quality
of food they are eating. Because type II diabetic patients have
significant issues with inflammatory tissue damage and fat that is
accumulating in all the wrong places, dietary supplements generally make
the process of improvement easier. For some people they make the process
possible. Choose a level of supplement support that makes sense to you.
Add to it, or adjust it, so as to help yourself stay on track. It is the
consistency of your entire program over a number of months that is your
path to getting better.

The following list of supplement ingredients is by no means complete, as
many other nutrients positively influence blood sugar and insulin. These
are nutrients I have used in clinical nutrition practice with
outstanding results. My point is to illustrate the many options and ways
nutrition can help support healthy blood sugar metabolism.

Nutrients, Supplements Herbs that have a positive influence on blood sugar and insulin:

DHA fish oil -It is now clear that patients with higher levels of DHA
have lower amounts of insulin resistance. DHA is known to reduce the
inflammation associated with blood sugar and insulin problems. It is
also clear that DHA directly boosts the production of adiponectin by
white adipose tissue, a hormone that may be key to the prevention of
type II diabetes.

Cinnamon -- Cinnamon is known to improve insulin resistance by
activating gene-related metabolic signaling. It individuals with
metabolic syndrome it has been shown to have improved fasting blood
glucose, systolic blood pressure, percentage body fat and increased lean
body mass. Cinnamon directly inhibits excess sugar from caramelizing
body cells and tissues (AGEs advanced glycation end products).

Banaba leaf --- The active component in banaba leaf, corosolic acid, has
been shown to support weight loss while lowering levels of blood sugar,
insulin, and triglycerides. This helped prevent fatty build up in the
liver while raising the very important level of diabetes-preventing
adiponectin. Banaba is a traditional remedy for diabetes in the
Philippines †and new studies are showing it to have a powerfully
helpful influence on white adipose tissue  a mechanism unlike any

Coffee berry Coffee is well known to decrease the incidence of type
II diabetes. Green coffee berries contain polyphenols and chlorogenic
acid, a nutrient that helps your liver metabolize sugar effectively by
modulating the enzyme glucose-6-phosphatase. It has been shown to
improve glucose tolerance, while reducing cholesterol and triglycerides.
It has also been shown to be effective for reducing mildly elevated
blood pressure.

Chromium Chromium helps blood sugar enter cells by supporting the
natural function of insulin at the cell membrane. The nutrient is
commonly lacking in individuals with type II diabetes. In type II
diabetic patients chromium is found to improve glucose, insulin,
cholesterol and Hemoglobin A1c.

Vanadium Vanadium is a mineral with insulin-like properties that has
been found to improve the metabolism of carbohydrates and fats, even in
diabetics. It helps promote better blood sugar metabolism in both muscle
and liver.

Inula racemosa  Inula has been found to increase the sensitivity of
insulin. In combination with Gymnema sylvestre it was found to guard
against steroid induced blood sugar problems, indicating that it helps
offset the effects of high stress on blood sugar metabolism.

Gymnema sylvestre Gymnema sylvestre is widely regarded as a top herb
to help combat the problem of type II diabetes. This herb has even been
shown to boost the number of pancreatic beta cells †a really unique
and helpful support nutrient.

Pterocarpus marsupium - Pterocarpus marsupium is a traditional remedy
for diabetes from India. It has insulin-like properties and it has a
regenerative effect on the beta cells of the pancreas (cells that make

Bitter melon Bitter melon is another Eastern traditional remedy for
diabetes, showing improved blood sugar and insulin function, improved
lipids, protection for beta cells, and reduced oxidative stress.

Grape seed extract - Grape seed extracts also turns down inflammation in
fat cells while boosting the highly protective adiponectin. They have
been shown to assist blood sugar entry to cells with insulin-like
activity. They also directly inhibit the advanced glycation end products
(AGEs) that form in the circulatory system from excess sugar.

Resveratrol - Resveratrol activates the SIRT1 gene, turning on other
genes that directly influence insulin function and fat burning, leading
researchers to conclude it may be helpful for diabetics. It reduces
inflammation in fat cells and boosts adiponectin, a key problem for

R-Alpha Lipoic Acid A nutrient that assists in carbohydrate
metabolism and insulin sensitivity, while acting as an antioxidant in
nerves to help diabetic neuropathy. A lack of ability to make lipoic
acid during aging increases inflammation and insulin resistance. Lipoic
acid also protects against the formation of AGEs.

Carnosine  Carnosine is a potent reducer of AGEs and can even reduce
the glycation of LDL cholesterol, leading researchers to conclude that
carnosine may have therapeutic potential in preventing diabetes-induced

LICORICE- FENNEL - These kill the appetite. That is also useful. It is
available in super markets, great in soup. Salads.

It is clear that nature's toolbox offers an impressive array of
nutrients that support healthy blood glucose and insulin metabolism.
While no person should ever consider these as a treatment in and of
themselves, they are a potent array of natural options that can be
plugged in, as desired, to an overall diet, exercise, and medical
treatment plan to help address the very difficult problem of type II

Summary Type II diabetes is at epidemic levels in America, reaching down
to teenagers and young adults in startling numbers. This problem
signifies that key regulatory systems have lost their natural balance
 meaning that homeostasis has been compromised. Unfortunately, this
locks in a path to poor quality health, the early onset of many serious
disease, and early death. The medical profession, relying on Big Pharma
drugs, has not been able to solve this problem and actually makes it
worse on a routine basis.

In order to make progress an overall program of lifestyle improvement
must be undertaken on a consistent basis. There are no shortcuts. There
is little margin for error. Weight must be lost in a gradual and
consistent way. Weight gain during any "treatment" means the "treatment"
is an utter failure. A healthy program requires making dietary
adjustments that have been explained in this article. Consistent
exercise is mandatory, the more the better. Dietary supplements can be
plugged into an overall program to help the program of recovery go
easier  and in some cases may be the key to getting the program on
track or keeping it from getting derailed. There are many options. Type
II diabetes can be prevented and in many people it can be completely

For a fully referenced version of this article:

About the author: Byron J. Richards, Board-Certified Clinical
Nutritionist, nationally-renowned nutrition expert, and founder of
Wellness Resources is a leader in advocating the value of dietary
supplements as a vital tool to maintain health. He is an outspoken
critic of government and Big Pharma efforts to deny access to natural
health products and has written extensively on the life-shortening and
health-damaging failures of the sickness industry.